polyclonal antibodies against pi3k Search Results


p pi3k  (Bioss)
95
Bioss p pi3k
P Pi3k, supplied by Bioss, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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phosphoinositide 3 kinase pi3k  (Danaher Inc)
86
Danaher Inc phosphoinositide 3 kinase pi3k
Phosphoinositide 3 Kinase Pi3k, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 86 stars, based on 1 article reviews
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antibodies against p pi3k  (Cell Signaling Technology Inc)
97
Cell Signaling Technology Inc antibodies against p pi3k
Fig. 5. PMLCPIR targets ITGB1 and evokes oncogenic <t>PI3K/AKT</t> signaling. (a) Schematic illustration of ITGB1/PI3K/AKT pathway and (b) stable knockdown of ITGB1 suppressed the PI3K/AKT pathway. (c) PMLCPIR overexpression evoked PI3K/AKT pathway, while knockdown inhibited PI3K/AKT pathway. (d) Knockdown of ITGB1 expression substantially rescued the activation of PI3K and AKT induced by PMLCPIR overexpression in HBE-T and A549 cells. (e) Knockdown of ITGB1 expression significantly rescued the effects of PMLCPIR overexpression in A549 cell migration and invasion. (f, g) Knockdown of ITGB1 expression significantly rescued the effects of PMLCPIR overexpression in A549 cell proliferation. Data are shown as means ± SD. *P<0.05, **P<0.01.
Antibodies Against P Pi3k, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/antibodies against p pi3k/product/Cell Signaling Technology Inc
Average 97 stars, based on 1 article reviews
antibodies against p pi3k - by Bioz Stars, 2026-03
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pi3 kinase p110 delta rabbit mab antibody  (ABclonal Biotechnology)
90
ABclonal Biotechnology pi3 kinase p110 delta rabbit mab antibody
Fig. 5. PMLCPIR targets ITGB1 and evokes oncogenic <t>PI3K/AKT</t> signaling. (a) Schematic illustration of ITGB1/PI3K/AKT pathway and (b) stable knockdown of ITGB1 suppressed the PI3K/AKT pathway. (c) PMLCPIR overexpression evoked PI3K/AKT pathway, while knockdown inhibited PI3K/AKT pathway. (d) Knockdown of ITGB1 expression substantially rescued the activation of PI3K and AKT induced by PMLCPIR overexpression in HBE-T and A549 cells. (e) Knockdown of ITGB1 expression significantly rescued the effects of PMLCPIR overexpression in A549 cell migration and invasion. (f, g) Knockdown of ITGB1 expression significantly rescued the effects of PMLCPIR overexpression in A549 cell proliferation. Data are shown as means ± SD. *P<0.05, **P<0.01.
Pi3 Kinase P110 Delta Rabbit Mab Antibody, supplied by ABclonal Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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pi3k p85  (Cell Signaling Technology Inc)
96
Cell Signaling Technology Inc pi3k p85
Primary antibodies used in this study.
Pi3k P85, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 96 stars, based on 1 article reviews
pi3k p85 - by Bioz Stars, 2026-03
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antibodies against pi3k  (Cell Signaling Technology Inc)
98
Cell Signaling Technology Inc antibodies against pi3k
Primary antibodies used in this study.
Antibodies Against Pi3k, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti-pi3k p110β  (Merck KGaA)
90
Merck KGaA anti-pi3k p110β
Primary antibodies used in this study.
Anti Pi3k P110β, supplied by Merck KGaA, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti p pi3k  (Bioss)
92
Bioss anti p pi3k
Primary antibodies used in this study.
Anti P Pi3k, supplied by Bioss, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti‑pi3k p85a  (Bioss)
94
Bioss anti‑pi3k p85a
Primary antibodies used in this study.
Anti‑Pi3k P85a, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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rabbit polyclonal anti p pi3k antibody  (Boster Bio)
92
Boster Bio rabbit polyclonal anti p pi3k antibody
Primary antibodies used in this study.
Rabbit Polyclonal Anti P Pi3k Antibody, supplied by Boster Bio, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti-rat pi3k  (Servicebio Inc)
90
Servicebio Inc anti-rat pi3k
Analysis of <t>PI3K,</t> AKT, and p-AKT expression levels by western blot and quantitative real-time PCR. (a) The blots showed that the SAH group had lower levels of PI3K, AKT, and p-AKT than the control group, and the graphs (b) show the ratio of expression compared with actin. BHD increased the expression of PI3K, AKT, and p-AKT as compared with the SAH group. (c) Quantitative real-time PCR analysis of PI3K, AKT, and p-AKT. Quantitative real-time PCR analysis of data was adjusted for GAPDH mRNA content ( n = 3) and examined in three separate quadruplicate experiments; data were shown as mean ± standard deviation ( n = 3); * P < 0.05, compared with the control group and # P < 0.05, compared with the SAH group, were used to indicate significance.
Anti Rat Pi3k, supplied by Servicebio Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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polyclonal pi3k p85α  (Santa Cruz Biotechnology)
96
Santa Cruz Biotechnology polyclonal pi3k p85α
Analysis of <t>PI3K,</t> AKT, and p-AKT expression levels by western blot and quantitative real-time PCR. (a) The blots showed that the SAH group had lower levels of PI3K, AKT, and p-AKT than the control group, and the graphs (b) show the ratio of expression compared with actin. BHD increased the expression of PI3K, AKT, and p-AKT as compared with the SAH group. (c) Quantitative real-time PCR analysis of PI3K, AKT, and p-AKT. Quantitative real-time PCR analysis of data was adjusted for GAPDH mRNA content ( n = 3) and examined in three separate quadruplicate experiments; data were shown as mean ± standard deviation ( n = 3); * P < 0.05, compared with the control group and # P < 0.05, compared with the SAH group, were used to indicate significance.
Polyclonal Pi3k P85α, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/polyclonal pi3k p85α/product/Santa Cruz Biotechnology
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Image Search Results


Fig. 5. PMLCPIR targets ITGB1 and evokes oncogenic PI3K/AKT signaling. (a) Schematic illustration of ITGB1/PI3K/AKT pathway and (b) stable knockdown of ITGB1 suppressed the PI3K/AKT pathway. (c) PMLCPIR overexpression evoked PI3K/AKT pathway, while knockdown inhibited PI3K/AKT pathway. (d) Knockdown of ITGB1 expression substantially rescued the activation of PI3K and AKT induced by PMLCPIR overexpression in HBE-T and A549 cells. (e) Knockdown of ITGB1 expression significantly rescued the effects of PMLCPIR overexpression in A549 cell migration and invasion. (f, g) Knockdown of ITGB1 expression significantly rescued the effects of PMLCPIR overexpression in A549 cell proliferation. Data are shown as means ± SD. *P<0.05, **P<0.01.

Journal: Journal of advanced research

Article Title: New insights into the function and mechanisms of piRNA PMLCPIR in promoting PM 2.5 -induced lung cancer.

doi: 10.1016/j.jare.2024.08.029

Figure Lengend Snippet: Fig. 5. PMLCPIR targets ITGB1 and evokes oncogenic PI3K/AKT signaling. (a) Schematic illustration of ITGB1/PI3K/AKT pathway and (b) stable knockdown of ITGB1 suppressed the PI3K/AKT pathway. (c) PMLCPIR overexpression evoked PI3K/AKT pathway, while knockdown inhibited PI3K/AKT pathway. (d) Knockdown of ITGB1 expression substantially rescued the activation of PI3K and AKT induced by PMLCPIR overexpression in HBE-T and A549 cells. (e) Knockdown of ITGB1 expression significantly rescued the effects of PMLCPIR overexpression in A549 cell migration and invasion. (f, g) Knockdown of ITGB1 expression significantly rescued the effects of PMLCPIR overexpression in A549 cell proliferation. Data are shown as means ± SD. *P<0.05, **P<0.01.

Article Snippet: Antibodies against P-PI3K (17366 s), P-AKT (4060 T), BCL2 (3498 T), and BAX (2772 s) were purchased from Cell Signaling Technology.

Techniques: Knockdown, Over Expression, Expressing, Activation Assay, Migration

Fig. 7. Inhibition of PMLCPIR diminished lung cancer growth in vivo. (a) Schematic of the construction of PMLCPIR inhibitor and control injection mice models. (b) The growth curves of tumor volume in anta-NC and anta-PMLCPIR group. (c) The representative images of the tumor in anta-NC and anta-PMLCPIR group. (d) The final tumor weight in anta-NC and anta-PMLCPIR groups. (e-f) The expression levels of PMLCPIR and ITGB1 in the tumors of anta-NC and anta-PMLCPIR group. (g) Representative images of immunohistochemical analyses (ki67 and ITGB1) in the tumors of anta-NC and anta-PMLCPIR group. (h) Western blotting analysis showing the expression levels of ITGB1/p- PI3K/AKT pathway in the tumors of anta-NC and anta-PMLCPIR group. Data are shown as means ± SD. *P<0.05, **P<0.01.

Journal: Journal of advanced research

Article Title: New insights into the function and mechanisms of piRNA PMLCPIR in promoting PM 2.5 -induced lung cancer.

doi: 10.1016/j.jare.2024.08.029

Figure Lengend Snippet: Fig. 7. Inhibition of PMLCPIR diminished lung cancer growth in vivo. (a) Schematic of the construction of PMLCPIR inhibitor and control injection mice models. (b) The growth curves of tumor volume in anta-NC and anta-PMLCPIR group. (c) The representative images of the tumor in anta-NC and anta-PMLCPIR group. (d) The final tumor weight in anta-NC and anta-PMLCPIR groups. (e-f) The expression levels of PMLCPIR and ITGB1 in the tumors of anta-NC and anta-PMLCPIR group. (g) Representative images of immunohistochemical analyses (ki67 and ITGB1) in the tumors of anta-NC and anta-PMLCPIR group. (h) Western blotting analysis showing the expression levels of ITGB1/p- PI3K/AKT pathway in the tumors of anta-NC and anta-PMLCPIR group. Data are shown as means ± SD. *P<0.05, **P<0.01.

Article Snippet: Antibodies against P-PI3K (17366 s), P-AKT (4060 T), BCL2 (3498 T), and BAX (2772 s) were purchased from Cell Signaling Technology.

Techniques: Inhibition, In Vivo, Control, Injection, Expressing, Immunohistochemical staining, Western Blot

Primary antibodies used in this study.

Journal: Frontiers in Aging Neuroscience

Article Title: Chronic cerebral hypoperfusion causes decrease of O-GlcNAcylation, hyperphosphorylation of tau and behavioral deficits in mice

doi: 10.3389/fnagi.2014.00010

Figure Lengend Snippet: Primary antibodies used in this study.

Article Snippet: PI3K p85 , Poly- , PI3K (p85) , , Cell Signaling Technology.

Techniques:

Analysis of PI3K, AKT, and p-AKT expression levels by western blot and quantitative real-time PCR. (a) The blots showed that the SAH group had lower levels of PI3K, AKT, and p-AKT than the control group, and the graphs (b) show the ratio of expression compared with actin. BHD increased the expression of PI3K, AKT, and p-AKT as compared with the SAH group. (c) Quantitative real-time PCR analysis of PI3K, AKT, and p-AKT. Quantitative real-time PCR analysis of data was adjusted for GAPDH mRNA content ( n = 3) and examined in three separate quadruplicate experiments; data were shown as mean ± standard deviation ( n = 3); * P < 0.05, compared with the control group and # P < 0.05, compared with the SAH group, were used to indicate significance.

Journal: Open Life Sciences

Article Title: BuyangHuanwu Decoction attenuates cerebral vasospasm caused by subarachnoid hemorrhage in rats via PI3K/AKT/eNOS axis

doi: 10.1515/biol-2022-0071

Figure Lengend Snippet: Analysis of PI3K, AKT, and p-AKT expression levels by western blot and quantitative real-time PCR. (a) The blots showed that the SAH group had lower levels of PI3K, AKT, and p-AKT than the control group, and the graphs (b) show the ratio of expression compared with actin. BHD increased the expression of PI3K, AKT, and p-AKT as compared with the SAH group. (c) Quantitative real-time PCR analysis of PI3K, AKT, and p-AKT. Quantitative real-time PCR analysis of data was adjusted for GAPDH mRNA content ( n = 3) and examined in three separate quadruplicate experiments; data were shown as mean ± standard deviation ( n = 3); * P < 0.05, compared with the control group and # P < 0.05, compared with the SAH group, were used to indicate significance.

Article Snippet: Rabbit polyclonal anti-rat PI3K (1:1000; Wuhan Servicebio Biotechnology Co., Ltd.), Akt, and p-AKT antibodies were used and anti-actin (monoclonal antibody, 1:1,000; Servicebio) served as a control.

Techniques: Expressing, Western Blot, Real-time Polymerase Chain Reaction, Control, Standard Deviation